UCHL1 provides diagnostic and antimetastatic strategies due to its deubiquitinating effect on HIF-1α

نویسندگان

  • Yoko Goto
  • Lihua Zeng
  • Chan Joo Yeom
  • Yuxi Zhu
  • Akiyo Morinibu
  • Kazumi Shinomiya
  • Minoru Kobayashi
  • Kiichi Hirota
  • Satoshi Itasaka
  • Michio Yoshimura
  • Keiji Tanimoto
  • Masae Torii
  • Terumasa Sowa
  • Toshi Menju
  • Makoto Sonobe
  • Hideaki Kakeya
  • Masakazu Toi
  • Hiroshi Date
  • Ester M. Hammond
  • Masahiro Hiraoka
  • Hiroshi Harada
چکیده

Hypoxia-inducible factor 1 (HIF-1) plays a role in tumour metastases; however, the genes that activate HIF-1 and subsequently promote metastases have yet to be identified. Here we show that Ubiquitin C-terminal hydrolase-L1 (UCHL1) abrogates the von Hippel-Lindau-mediated ubiquitination of HIF-1α, the regulatory subunit of HIF-1, and consequently promotes metastasis. The aberrant overexpression of UCHL1 facilitates distant tumour metastases in a HIF-1-dependent manner in murine models of pulmonary metastasis. Meanwhile, blockade of the UCHL1-HIF-1 axis suppresses the formation of metastatic tumours. The expression levels of UCHL1 correlate with those of HIF-1α and are strongly associated with the poor prognosis of breast and lung cancer patients. These results indicate that UCHL1 promotes metastases as a deubiquitinating enzyme for HIF-1α, which justifies exploiting it as a prognostic marker and therapeutic target of cancers.

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2015